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Every time your cells divide, the "telomeres," or caps on the ends of your chromosomes, get a little shorter. Elderly creatures begin to suffer diseases of old age partly because their telomeres have become so short, and cellular division becomes difficult and error-prone. That's why telomeres are the subject of intensive study: If we could figure out the mechanism that keeps our telomeres from shortening, it's possible we could prolong our lives. Now a group of California researchers have discovered that a protein called Est3 (pictured) is partly responsible for keeping telomeres long. Manipulating that protein could bring us one step closer to healthy cell longevity. More »
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